In contrast to neurodegerative conditions and cancer, the contribution of TG2 activity to celiac sprue pathogenesis has been relatively effectively founded. The deamidation of particular glutamine residues targeted by TG2 in gluten peptides is demonstrated to boost peptide binding for the sickness-related proteins HLA-DQ2/8, that are the one known genetic https://ldn-27219-pharmacological13568.gynoblog.com/30923721/ldn-27219-preclinical-research-for-dummies