Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. Then again, our capability to rescue SERT operate in KI midbrain synaptoneurosomes by the inhibition of FAK suggests elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by greater pFAK localization in five-HT synapses. Our https://pro33login34566.onesmablog.com/pro33-an-overview-72797090